We are a diverse group of scientists and surgeons who seek to answer the questions—why would muscle turn into fat, and can this phenomenon be leveraged to promote muscle regeneration following musculoskeletal injury and repair?
Our team studies how chromatin receives, processes, and amplifies environmental stress signals that drive changes in cell states leading to heart disease.
Our lab leverages cutting-edge machine learning and experimental genomics to map the gene regulatory networks disrupted in cardiovascular disease and discover network-correcting therapeutics.
The research interest of my lab lies at the intersection of CRISPR-based genome editing and protein/cell engineering with special focus on hematopoietic stem cells and red blood cell disorders.
